How aids are spreaded?


Answers:    well, its a very intelligent ques u hav asked.
vectors are- breast milk, blood, semen vaginal fluid... nd othr body fluids (except saliva)
it is transmitted thru unprotected sex wid an infected partner (or whn u hav worn a condom bt latex breaks), sharing the same syringe (one whoz injected wid a syringe tht be used earlier in the body of an HIV+ gets the virus), n from the infected mother to the child contained by her womb...
the methods of protection are- always use a condom, be faithful to ur partner so tht there's no risk of carrying HIV in any of the two, always use disposable syringes in medical procedures (and if they are not available, sterilise the syringe so tht the virus gets destroyed)....
a such, there's no pills of AIDS available in any country of the world, bt the doctors' teams are constantly working towards the success... demise is a surity in the case of AIDS bt aftr yrs of research, the doctors hav developed a medicine which is fit of delaying the death... the immunity of the body is totally destroyed within the presence of HI Virus... the person falls a prey to evry big and small medical problem..... it can b detected thru Eliza, Western Blot and Chain Polymer test... Eliza and chain polymer trial are the primary tests bt western blot is a sure-shot test...
care!
Cause
For more details on this topic, see HIV.

Scanning electron micrograph of HIV-1, colored green, budding from a cultured lymphocyte.AIDS is the most severe acceleration of infection with HIV. HIV is a retrovirus that primarily infects necessary organs of the human immune system such as CD4+ T cells (a subset of T cells), macrophages and dendritic cells. It directly and indirectly destroys CD4+ T cells.[34]
HIV have killed so many CD4+ T cells that within are fewer than 200 of these cells per microliter (uL) of blood, cellular immunity is lost. Acute HIV infection progresses over time to clinical hidden HIV infection and then to early symptomatic HIV infection and later to AIDS, which is identified any on the basis of the amount of CD4+ T cells remaining in the blood, and/or the presence of undisputed infections, as noted above.[35]

In the absence of antiretroviral therapy, the median time of progression from HIV infection to AIDS is nine to ten years, and the median survival time after developing AIDS is only 9.2 months.[36] However, the rate of clinical disease progression vary widely between individuals, from two weeks up to 20 years.

Poor access to robustness care and the existence of coexisting infections such as tuberculosis also may predispose people to faster disease progression.[36][39][40] The infected person's genetic inheritance plays an important role and some population are resistant to certain strains of HIV. An example of this is people with the homozygous CCR5-Δ32 flux are resistant to infection with certain strains of HIV.[41] HIV is genetically variable and exists as different strains, which raison d`être different rates of clinical disease progression.[42][43][44]


al transmission occurs beside the contact between sexual secretions of one person with the rectal, genital or oral mucous membranes of another. Unprotected open sexual acts are riskier than unprotected insertive sexual acts, and the risk for transmitting HIV through unprotected anal intercourse is greater than the risk from vaginal intercourse or oral sex.

However, oral sex is not entirely safe, as HIV can be transmitted through both insertive and amenable oral sex.[45][46] Sexual assault greatly increases the risk of HIV transmission as condoms are rarely employed and physical trauma to the vagina occurs frequently, facilitate the transmission of HIV.[47]

Other sexually transmitted infections (STI) increase the risk of HIV transmission and infection, because they cause the disruption of the common epithelial barrier by genital ulceration and/or microulceration; and by accumulation of pools of HIV-susceptible or HIV-infected cells (lymphocytes and macrophages) surrounded by semen and vaginal secretions. Epidemiological studies from sub-Saharan Africa, Europe and North America suggest that genital ulcers, such as those caused by syphilis and/or chancroid, increase the risk of becoming infected near HIV by about fourfold. There is also a significant althoug
It is spread through contact with an infected persons blood or sexual fluids. It can not be caught through saliva


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