Can HPV fully clear from the body or not?

I've been researching HPV on the internet - and it appears that there is much controversy over whether HPV can fully be eradicated from the body by the immune system.
what I've read, it seems most inhabitants believe that the virus does completely leave the body after 2 years or so, in most cases - with a sound immune system. -- On the other hand, I've read that the virus becomes "dormant" after some time, and stays with you for enthusiasm without symptoms (in most cases, but symptoms do reoccur in some occasions).

-- One of the points that I've read people formulate about why it stays with you for life is "HPV is a virus - and virus stay next to you for life no matter what" --- does that mean STOMACH VIRUS'S stay near you for life too?? That doesn't make any sense.

Within 2 years, the virus is usually reduced to indetectable levels. It's indetectable, but is it fully gone? No one can speak about you.
We can acquire new HPV near a new HPV type is possible with a new sex partner...also the virus can reactivate years after the initial infection. The body can build cell imperviousness to an acquired HPV type...but it may not...years later showing abnormal cell change...

There is no cure for the tell future sex partners of your ancient HPV infection....
e this will help

The virus takes up residence in our cell and alter the properties of that cell…the cell is transformed…the virus can stay in a sleeping state or does not replicated due to the cell me immunity.

In an article published in the Proceedings of the National Academy of Sciences in March, the research squad reports that a viral replication protein known as E2 binds the circular viral DNA to cell structures called spindle fibers that are present in a cell when it divides, a process specified as mitosis. In mitosis, a single cell divides in two, creating two genetically identical daughter cells. By latching onto the spindle fibers of the cell as it divides, HPV DNA also divides and replicates itself within each of the new daughter cells where on earth it can continue to replicate and persist indefinitely.

“In effect, HPV is able to mimic our own chromosomes, behave as a sort of ‘mini-chromosome’, independently replicating and keeping pace as the cellular chromosomes replicate and the cell divides,” says Tom Broker, PhD, professor of biochemistry and molecular genetics and co-author of the composition. “This allows the virus to remain in our bodies indefinitely, with the potential of causing serious disease years, even decades, after first exposure.

Following infection, HPV enter a period of quiescence that lasts about 2-12 months. Most HPV infections resolve minus symptoms (subclinical) at this stage, presumably due to the emergence of the host’s cell-mediated immune response beginning approximately 3 months after infection (Figure 17). This immune response either eradicates the virus or suppresses it to non-detectable levels. Therefore, it is not however known whether an HPV infection that appears to have cleared clinically is really eradicated or simply remains below the sensitivity level for detection next to current molecular techniques.
Some HPV infections are thought to be suppressed and their genomes maintained in a long-term sleeping state (i.e., subclinical infection with a very small group of cells presumably maintain infection at low DNA copy numbers). Support for a latent state for HPV infection comes from the observation that in some women genital wart can resolve spontaneously only to recur (i.e., reactivate) during pregnancy or when the immune system becomes compromised (e.g., HIV infection). It is not yet clear how commonly latency occur in immunocompetent hosts, its ultimate duration, the circumstances and mechanisms that trigger re-emergence of HPV into a detectable state, whether undeveloped HPV infection is ultimately eliminated in most individuals, or whether latent infection can carry on, possibly leading to cervical cancer.
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Mechanism of genomic instability in cells infected near the high-risk human papillomaviruses.
Kadaja M, Isok-Paas H, Laos T, Ustav E, Ustav M.
PV-related cancers, the "high-risk" human papillomaviruses (HPVs) are frequently found integrated into the cellular genome. The integrated subgenomic HPV fragments express viral oncoproteins and carry an origin of DNA replication to be exact capable of initiating bidirectional DNA re-replication in the presence of HPV replication proteins E1 and E2, which ultimately leads to rearrangements in the locus of the integrated viral DNA. The current study indicates that the E1- and E2-dependent DNA replication from the integrated HPV origin follows the "onion skin"-type replication mode and generates a heterogeneous population of replication intermediates. These include linear, branched, open circular, and supercoiled plasmids, as identified by two-dimensional neutral-neutral gel-electrophoresis. We used immunofluorescence analysis to show that the DNA repair/recombination centers are assembled at the sites of the integrated HPV replication. These centers conscript viral and cellular replication proteins, the MRE complex, Ku70/80, ATM, Chk2, and, to some extent, ATRIP and Chk1 (S317). In addition, the synthesis of histone gammaH2AX, which is a hallmark of DNA double strand breaks, is induced, and Chk2 is activated by phosphorylation in the HPV-replicating cell. These changes suggest that the integrated HPV replication intermediates are processed by the activated cellular DNA repair/recombina
Once you contract a virus it is there forever and you can build antibodies to a virus however, this virus attacks and the change that occur can result in cancer.
Unofrtunetly it does stay but i do know what you mean as in not adjectives viruses stay

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